The Mammalian Stress Mechanism Explains Covid, Long Covid and Sudden Death
Abstract:
Mammalian stress mechanism (MSM) hyperactivity explains the bewildering local and systemic manifestations of coronavirus infestations. The “normal” coronavirus attacks the pulmonary endothelium and causes mild and transient localized MSM hyperactivity that exaggerates exudates which cause coughing and sneezing that spreads the contagion. It also exaggerates the “leakage” of pulmonary tissue factor into systemic circulation, causing mild systemic MSM hyperactivity that explains the fever, fatigue, and malaise of the common cold. The exaggerated virulence of the “novel” coronavirus causes severe pulmonary MSM hyperactivity, which generates excessive exudates that disrupt gas exchange in the lung and manifests as ARDS (acute respiratory distress syndrome). It also causes extreme leakage of pulmonary tissue factor into systemic circulation, causing severe systemic MSM hyperactivity that manifests as loss of smell, taste and hair as well as fever, fatigue, and malaise. Both normal and novel coronaviruses remain confined to the lung, but the mRNA “immunization” introduces the coronavirus “spike protein” directly into systemic circulation, where it proliferates and attacks the vascular endothelium throughout the body. The resulting systemic MSM hyperactivity secondarily elevates nonspecific immune activity but fails to elicit specific immune activity that conveys lasting protection. Meanwhile, the systemic disruption of the vascular endothelium causes pericarditis, myocarditis, and infertility, as well as fever, fatigue, malaise, and loss of smell, taste, and hair that mimics COVID illness. In addition, it causes dangerous hypercoagulability of blood that disrupts oxygen transport and delivery, causing sudden, unexpected death in some healthy young individuals and lingering “Long COVID” syndrome in others.
